Urticaria is a common disorder; learn more on the pathophysiology of hives and/or angioedema, usual triggers and differential diagnoses. Trigger avoidance and second-generation antihistamines are the cornerstone of management
The skin deep conversation – focus on urticaria and angioedema
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- Question 1 of 15
1. Question
1. The co-occurrence of both wheals and angioedema is seen in __% of cases of urticaria?
- Question 2 of 15
2. Question
2. Urticaria is generally classified according to:
- Question 3 of 15
3. Question
3. Which statement is false? Acute urticaria refers to wheals with or without angioedema:
- Question 4 of 15
4. Question
4. Which statement is false? Angioedema:
- Question 5 of 15
5. Question
5. Mast cell degranulation leads to the rapid release of which inflammatory mediators?
- Question 6 of 15
6. Question
6. Inflammation-mediated mechanisms for itch, wheals and angioedema include:
- Question 7 of 15
7. Question
7. One proposed mechanism for the pathogenesis of chronic urticaria involves the development of IgG autoantibodies to IgE or high-affinity IgE receptors on both mast cells and basophils:
- Question 8 of 15
8. Question
8. Which medications are commonly known to cause urticaria with or without angioedema?
- Question 9 of 15
9. Question
9. Which statement is incorrect? Chronic urticaria:
- Question 10 of 15
10. Question
10. Triggers or aggravating factors can be identified in __% of children with chronic urticaria?
- Question 11 of 15
11. Question
11. Autoimmunity is present in __% of patients with CSU?
- Question 12 of 15
12. Question
12. Pseudoallergens that promote inflammation or reduce anti-inflammatory mechanisms may predispose susceptible individuals to CSU:
- Question 13 of 15
13. Question
13. Which statement is incorrect?
- Question 14 of 15
14. Question
14. Which statement is incorrect? For patients with CSU:
- Question 15 of 15
15. Question
15. Second-generation antihistamines induce fewer CNS-related side effects than first-generation antihistamines, due to poor CNS penetration:
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